Clinical form of Lyme Arthritis Disease

The clinical form of Lyme disease - Lime Arthritis - has a tendency to chronic and recurrent flow. Lyme disease is a multisystem disease that is often called a "new great imitator" in connection with a variety of clinical manifestations resembling syphilis. It is a complex immuno-mediated disease, infectious by origin and inflammatory or "rheumatic" in essence. These signs make Lyme disease a unique model for studying the infectious etiology of rheumatic diseases. Lime arthritis was identified as a clinical manifestation in the late 1970s after the outbreak of mono and oligoarthicular arthritis in children with Lyme disease in Connecticut, USA. Today it is known that the defeat of the joints is a late manifestation of Lyme disease. However, some patients may report cases of tick bite or the presence of migratory erythema.

Epidemiology. Pathogenesis.

Complaints of skeletal-muscular discomfort cause patients to contact doctors. In particular, the transitional episodes of spontaneous synovitis of the knee joints (often monosinovite) are common in the early stages of the progression of Lyme disease, and if the treatment does not conduct, in 60% of patients developing Lime arthritis. Arthritis manifestations are quickly responding to antibiotics therapy, and this is a reason to consider it necessary to include Lyme disease in differential diagnosis as a potential cause of spontaneous synovitis, which, in turn, can prevent the development of heavier symptoms related to the disease.

Thus, esophageal damage in rheumatic diseases - frequent and clinically and prognostically significant manifestations of systemic connecting pathology. The formation of esophageal complications in patients with the above nosologies occurs under the influence of many factors in a certain interaction among themselves. However, the fundamental basis of the damage to the esophagus is autoimmune inflammation and the hyperproduction of the connective tissue is caused by it.
The mysterious feature of Lyme disease is the slow increase in symptoms from the musculoskeletal system, which can last after the total course of treatment, and some patients develop chronic inflammatory arthritis, resistant to antibiotic therapy. These experimental studies indicate that Borrelia Burgdorferi antigens remain in cartilage for a long period after etiological therapy and contribute to stimulation of the synthesis of the necrosis factor of tumor - from macrophages, which leads to the chronization of the process in the joints. Best Training Courses in South Africa on https://southafricaskills.com/ . Find best training providers in South Africa.